The histone deacetylase inhibitor valproic acid selectively induces proteasomal degradation of HDAC2

Oliver H. Krämer, Ping Zhu, Heather P. Ostendorff, Martin Golebiewski, Jens Tiefenbach, Marvin A. Peters, Boris Brill, Bernd Groner, Ingolf Bach, Thorsten Heinzel, Martin Göttlicher

Author Affiliations

  1. Oliver H. Krämer1,4,
  2. Ping Zhu2,4,
  3. Heather P. Ostendorff3,
  4. Martin Golebiewski2,
  5. Jens Tiefenbach1,
  6. Marvin A. Peters3,
  7. Boris Brill1,
  8. Bernd Groner1,
  9. Ingolf Bach3,
  10. Thorsten Heinzel*,1 and
  11. Martin Göttlicher*,2,4
  1. 1 Georg‐Speyer‐Haus, Paul‐Ehrlich‐Straße 42‐44, D‐60596, Frankfurt, Germany
  2. 2 Forschungszentrum Karlsruhe, Institute of Toxicology and Genetics, H.‐v.‐H.‐Platz 1, D‐76344, Eggenstein, Germany
  3. 3 Zentrum für Molekulare Neurobiologie (ZMNH), Universität Hamburg, Martinistraße 85, D‐20251, Hamburg, Germany
  4. 4 Present address: GSF National Research Center on Environment and Health, Institute of Toxicology, Ingolstädter Landstraße 1, D‐85754, Neuherberg, Germany
  1. *Corresponding authors. E-mail: heinzel{at} or E-mail: martin.goettlicher{at}
  1. O.H.Krämer and P.Zhu contributed equally to this work

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